Stanford Study: Beta-Amyloid Protein Fragment May Be an Impetus for Alzheimer’s

Scientists at the Stanford University School of Medicine have demonstrated how the beta-amyloid protein fragment destroys synapses and clumps into plaque that kills cells, leading to Alzheimer’s disease. “Our discovery suggests that Alzheimer’s disease starts to manifest long before plaque formation becomes evident,” says the senior author of the study. The study was conducted on mice and in human brain tissue, and may help to assess the failures of many large-scale  attempts to slow the disease’s progression. The findings may also lead to ways to treat Alzheimer’s at its early stages. Click here to read the full article.